Difference between revisions of "Shock"
(Created page with 'File:lighterstill.jpgright|frame ==Origin== Middle French choc, from choquer to strike against, from Old French choquier, probably of Germanic ...') |
m (Text replacement - "http://" to "https://") |
||
| Line 3: | Line 3: | ||
==Origin== | ==Origin== | ||
Middle French choc, from choquer to strike against, from Old French choquier, probably of Germanic origin; akin to Middle Dutch schocken to jolt | Middle French choc, from choquer to strike against, from Old French choquier, probably of Germanic origin; akin to Middle Dutch schocken to jolt | ||
| − | *[ | + | *[https://en.wikipedia.org/wiki/16th_century 1565] |
==Definitions== | ==Definitions== | ||
*1: the impact or encounter of [[individuals]] or [[groups]] in combat | *1: the impact or encounter of [[individuals]] or [[groups]] in combat | ||
| Line 16: | Line 16: | ||
Acute [[stress]] [[reaction]] (also called acute stress disorder, psychological '''shock''', mental shock, or simply, shock) is a [[psychological]] condition arising in [[response]] to a terrifying or traumatic [[event]]. It should not be [[confused]] with the unrelated circulatory condition of shock. | Acute [[stress]] [[reaction]] (also called acute stress disorder, psychological '''shock''', mental shock, or simply, shock) is a [[psychological]] condition arising in [[response]] to a terrifying or traumatic [[event]]. It should not be [[confused]] with the unrelated circulatory condition of shock. | ||
| − | "Acute stress response" was first described by [ | + | "Acute stress response" was first described by [https://en.wikipedia.org/wiki/Walter_Cannon Walter Cannon] in the 1920s as a [[theory]] that [[animals]] react to threats with a general discharge of the sympathetic [[nervous system]]. The [[response]] was later recognized as the first stage of a general adaptation syndrome that regulates stress responses among vertebrates and other [[organisms]]. |
| − | The onset of a [[stress]] response is associated with specific physiological [[actions]] in the sympathetic [[nervous system]], both directly and indirectly through the release of [ | + | The onset of a [[stress]] response is associated with specific physiological [[actions]] in the sympathetic [[nervous system]], both directly and indirectly through the release of [https://en.wikipedia.org/wiki/Epinephrine epinephrine] and to a lesser extent [https://en.wikipedia.org/wiki/Norepinephrine norepinephrine] from the [https://en.wikipedia.org/wiki/Adrenal_medulla medulla] of the [https://en.wikipedia.org/wiki/Adrenal_glands adrenal glands]. The release is triggered by [https://en.wikipedia.org/wiki/Acetylcholine acetylcholine] released from pre-[https://en.wikipedia.org/wiki/Ganglion ganglionic] sympathetic nerves. These catecholamine [[hormones]] facilitate immediate [[physical]] reactions by triggering increases in heart rate and [[breathing]], constricting blood vessels in many parts of the [[body]]—but not in muscles (vasodilation), brain, lungs, and heart—and tightening muscles. An [[abundance]] of catecholamines at neuroreceptor sites facilitates reliance on [[spontaneous]] or [[intuitive]] [[behaviors]] often related to combat or [[escape]]. |
| − | Normally, when a [[person]] is in a serene, unstimulated state, the "firing" of [[neurons]] in the [ | + | Normally, when a [[person]] is in a serene, unstimulated state, the "firing" of [[neurons]] in the [https://en.wikipedia.org/wiki/Locus_ceruleus locus ceruleus] is minimal. A novel stimulus, once perceived, is relayed from the [https://en.wikipedia.org/wiki/Sensory_cortex sensory cortex] of the [[brain]] through the [https://en.wikipedia.org/wiki/Thalamus thalamus] to the [https://en.wikipedia.org/wiki/Brain_stem brain stem]. That route of signaling increases the rate of noradrenergic activity in the locus ceruleus, and the [[person]] becomes alert and [[attentive]] to the [[environment]]. |
| − | If a [[stimulus]] is perceived as a threat, a more [[intense]] and prolonged discharge of the locus ceruleus activates the sympathetic division of the [ | + | If a [[stimulus]] is perceived as a threat, a more [[intense]] and prolonged discharge of the locus ceruleus activates the sympathetic division of the [https://en.wikipedia.org/wiki/Autonomic_nervous_system autonomic nervous system] (Thase & Howland, 1995). The activation of the sympathetic [[nervous system]] leads to the release of norepinephrine from nerve endings acting on the [[heart]], blood vessels, respiratory centers, and other sites. The ensuing physiological changes constitute a major part of the acute stress response. The other major player in the acute stress response is the [https://en.wikipedia.org/wiki/Hypothalamic-pituitary-adrenal_axis hypothalamic-pituitary-adrenal axis].[https://en.wikipedia.org/wiki/Acute_stress_reaction] |
[[Category: Health]] | [[Category: Health]] | ||
[[Category: Biology]] | [[Category: Biology]] | ||
Latest revision as of 02:36, 13 December 2020
Origin
Middle French choc, from choquer to strike against, from Old French choquier, probably of Germanic origin; akin to Middle Dutch schocken to jolt
Definitions
- 1: the impact or encounter of individuals or groups in combat
- 2a : a violent shake or jar : concussion
- 3a (1) : a disturbance in the equilibrium or permanence of something (2) : a sudden or violent mental or emotional disturbance
- b : something that causes such disturbance <the loss came as a shock>
- c : a state of being so disturbed <were in shock after they heard the news>
- 4: a state of profound depression of the vital processes associated with reduced blood volume and pressure and caused usually by severe especially crushing injuries, hemorrhage, or burns
- 5: sudden stimulation of the nerves and convulsive contraction of the muscles caused by the discharge of electricity through the animal body
Description
Acute stress reaction (also called acute stress disorder, psychological shock, mental shock, or simply, shock) is a psychological condition arising in response to a terrifying or traumatic event. It should not be confused with the unrelated circulatory condition of shock.
"Acute stress response" was first described by Walter Cannon in the 1920s as a theory that animals react to threats with a general discharge of the sympathetic nervous system. The response was later recognized as the first stage of a general adaptation syndrome that regulates stress responses among vertebrates and other organisms.
The onset of a stress response is associated with specific physiological actions in the sympathetic nervous system, both directly and indirectly through the release of epinephrine and to a lesser extent norepinephrine from the medulla of the adrenal glands. The release is triggered by acetylcholine released from pre-ganglionic sympathetic nerves. These catecholamine hormones facilitate immediate physical reactions by triggering increases in heart rate and breathing, constricting blood vessels in many parts of the body—but not in muscles (vasodilation), brain, lungs, and heart—and tightening muscles. An abundance of catecholamines at neuroreceptor sites facilitates reliance on spontaneous or intuitive behaviors often related to combat or escape.
Normally, when a person is in a serene, unstimulated state, the "firing" of neurons in the locus ceruleus is minimal. A novel stimulus, once perceived, is relayed from the sensory cortex of the brain through the thalamus to the brain stem. That route of signaling increases the rate of noradrenergic activity in the locus ceruleus, and the person becomes alert and attentive to the environment.
If a stimulus is perceived as a threat, a more intense and prolonged discharge of the locus ceruleus activates the sympathetic division of the autonomic nervous system (Thase & Howland, 1995). The activation of the sympathetic nervous system leads to the release of norepinephrine from nerve endings acting on the heart, blood vessels, respiratory centers, and other sites. The ensuing physiological changes constitute a major part of the acute stress response. The other major player in the acute stress response is the hypothalamic-pituitary-adrenal axis.[1]
